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81. Scherrer U, Vollenweider L, Delabays A, Savcic M, Eichenberger U, Kleger G-R, Fikrle A, Ballmer PE, Nicod P, and Bartsch P. Inhaled nitric oxide for high-altitude pulmonary edema. N. Engl. J. Med. 1996; 334: 624-630. 82. Secomb TW and Pries AR. Information transfer in microvascular networks. Microcirculation 2002; 9: 377-387. 83. Shirai M, Sada K, and Ninomiya I. Effects of regional alveolar hypoxia and hypercapnia in small pulmonary vessels in cats. J. Appl. Physiol. 1986; 61: 440-448. 32 84.

Hypoxic contraction of cultured pulmonary vascular smooth muscle cells. Am. J. Respir. Cell. Mol. Biol. 1990; 3: 457-465. 33. Nagasaka Y, Bhattacharya J, Nanjo S, Cropper MA, and Staub NC. Micropuncture 14 measurement of lung microvascular pressure profile during hypoxia in cats. Circ. Res. 1984; 54: 90-95. 34. Rotta A. Physiologic condition of the heart in the natives of high altitudes. Am. Heart J. 1947; 33: 669-676. 35. Rudolph AM. Fetal and neonatal pulmonary circulation. Annu. Rev. 1979; 41: 383­ 395.

5) that the vessel size dependence in hypoxia reactivity of isolated pulmonary arterial rings was correlated with the relative distributions of three electro­ physiologically distinct smooth muscle cell phenotypes. As indicated in other chapters, pulmonary arterial smooth muscle cells have become the model system for studying the mechanisms of HPV. However, normal pulmonary arteries smaller than about in diameter, which have few if any smooth muscle cells, constrict in response to hypoxia as well as the larger vessels from which smooth muscle cells are normally harvested (37, 92).

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